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(30137) pCAX APP 695

Supplied by Addgene Inc, cited in 25 publications.

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25 citations

General Information

Supplier
Addgene Inc
Type
Plasmid

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Expression Systems

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25 citations

found in the literature

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β-Amyloid and Glutathione Dysregulation Cooperatively Drive Lipid Peroxidation and Ferroptosis in Neuron-Like Cells

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Preprint on BioRxiv : the Preprint Server for Biology on 17 April 2026 by Radeen, K. R., Hao, C., et al.

Amyloid-β protein precursor modulates mitophagy and mitochondrial function through its cellular localization.

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In Journal of Alzheimer's Disease : JAD on 1 September 2025 by Strope, T. A., Troutwine, B., et al.

Familial Alzheimer's disease mutations in amyloid precursor protein impair calcineurin signaling to NMDA receptors.

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In The Journal of Biological Chemistry on 1 February 2025 by Tavalin, S. J.

Plasma membrane repair defect in Alzheimer's disease neurons is driven by the reduced dysferlin expression.

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In The FASEB Journal on 31 October 2024 by Bulgart, H. R., Lopez Perez, M. A., et al.

Amyloid-β-Induced Dendritic Spine Elimination Requires Ca2+-Permeable AMPA Receptors, AKAP-Calcineurin-NFAT Signaling, and the NFAT Target Gene Mdm2.

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In ENeuro on 1 March 2024 by Martinez, T. P., Larsen, M. E., et al.

Extracellular disposal of nuclear waste by APP: a protective mechanism impaired in Alzheimer’s disease

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Preprint on BioRxiv : the Preprint Server for Biology on 12 February 2024 by Dougnon, G., Otsuka, T., et al.

Inhibiting USP16 rescues stem cell aging and memory in an Alzheimer's model.

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In eLife on 21 March 2022 by Reinitz, F., Chen, E. Y., et al.

EDEM1 Regulates Amyloid Precursor Protein (APP) Metabolism and Amyloid-β Production.

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In International Journal of Molecular Sciences on 23 December 2021 by Nowakowska-Gołacka, J., Czapiewska, J., et al.

Mitochondrial Membrane Potential Influences Amyloid-β Protein Precursor Localization and Amyloid-β Secretion.

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In Journal of Alzheimer's Disease : JAD on 23 November 2021 by Wilkins, H. M., Troutwine, B. R., et al.

Amyloid precursor protein is a restriction factor that protects against Zika virus infection in mammalian brains.

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In The Journal of Biological Chemistry on 11 December 2020 by Lingel, A., Lin, H., et al.

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